An anatomical and metabolic interface between the mother and the fetus, villous placenta (placental disc) is of the utmost importance for the well-being of the fetus. In this context, chronic

2020-03-01

Even if the classic intranuclear and intracytoplasmic inclusions are not seen, an immunohistochemical study for CMV can be beneficial, as it will highlight infected cells and eosinophilic debris within the villi. Objective . To elucidate differences in the frequency and severity of acute chorioamnionitis (CAM) and chronic villitis in placentas from stillborns compared with liveborns at term and to evaluate other risk factors and placental findings. Design . Case-control study. Setting . All delivery wards in major Stockholm area.

Villitis placenta

In case of villitis, polymerase chain reaction (PCR) on influenza virus was performed on placental tissue. RESULTS: 29 patients had influenza infection. Placentas of 15 of these patients were collected and examined. villitis include lymphohistiocytic, lymphocytic, lympho-plasmocytic lesions, and granulomatous inﬂammation with multinucleate giant cells [1–5] (Fig. 1). The percentage of placentas with chronic villitis is reported as being 7, 6–10% intheUnitedStates[1,3],13.6%intheUnitedKingdom[2], 14.2%inNewZealand[5]andupto33.8%inArgentina[4].

The placentas of patients with influenza infection were examined for histologic signs of chronic villitis. In case of villitis, polymerase chain reaction (PCR) on influenza virus was performed on placental tissue. Results . 29 patients had influenza infection. Placentas of 15 of these patients were collected and examined.

Placenta; Villitis of unknown etiology Summary Villitis of unknown etiology (VUE) is an important pattern of placental injury occurring predominantly in term placentas. Although overlapping with infectious villitis, its clinical and histologic characteristics are distinct. It is a common lesion, affecting 5% to 15% of all placentas. When low-grade Chronic villitis was observed in 4% of placentas examined after delivery between 22 and 28 weeks’ gestation, 9–14% between 29 and 32 weeks’ gestation and 16–44% between 33 and 36 weeks’ gestation [29,36].

(4) The increased maternal serum level and enhanced placental expression of TNF-α could represent the underlying mechanism for the development of villitis

Placentas of 15 of these patients were collected and examined. villitis include lymphohistiocytic, lymphocytic, lympho-plasmocytic lesions, and granulomatous inﬂammation with multinucleate giant cells [1–5] (Fig. 1). The percentage of placentas with chronic villitis is reported as being 7, 6–10% intheUnitedStates[1,3],13.6%intheUnitedKingdom[2], 14.2%inNewZealand[5]andupto33.8%inArgentina[4]. RESULTS: 178 placentas were affected (78 cases of CVUE, 24 cases of CIUE and 76 cases of combined lesions involving both villitis and intervillositis) including 12 cases of recurrence.

In most cases, the inflammation occurs in a term placenta. VUE is seen in 5-15% of the placentas The placenta is an organ that connects the developing fetus to the uterine wall. Placenta; Villitis of unknown etiology Summary Villitis of unknown etiology (VUE) is an important pattern of placental injury occurring predominantly in term placentas. Although overlapping with infectious villitis, its clinical and histologic characteristics are distinct. It is a common lesion, affecting 5% to 15% of all placentas. When low-grade Chronic villitis was observed in 4% of placentas examined after delivery between 22 and 28 weeks’ gestation, 9–14% between 29 and 32 weeks’ gestation and 16–44% between 33 and 36 weeks’ gestation [29,36].
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Chronic intervillositis is a less The origins of placental dysfunction are poorly understood.

Acute Villitis of Placenta is a bacterial infection of the chorionic villi The placenta is an organ that connects the developing fetus to the uterine wall.
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Placenta. Nonneoplastic placental conditions and abnormalities. Other associated lesions may include atherosis, acute chorioamnionitis and rarely chronic villitis

On examining the junction between the fused dichorionic placentas, Villitis was studied in placentas from 445 singleton infants from an ethnically homogeneous population with a good socioeconomic standard. There were 161 infants small for gestational age (SGA) and 284 appropriate for gestational age (AGA). Villitis was found in 12 SGA‐placentas (7.5 per cent) and 8 AGA placentas (2.8 per cent) (p < 0.05).